Mystery Vitamin C: Strange Interaction With Ebola and Other Oddities
The information presented in this article will probably shock you.
Before we get more deep into things it is important to note that refined white sugar is used to manufacture vitamin C. Therefore, it’s not surprising that the vitamin C molecule closely resembles the sugar molecule.
Your body has the ability to convert vitamin C back into refined, white sugar and use it as fuel and this fuel is able to cross the blood–brain barrier, which blocks many nutrients.
In a survival situation, when your body needs usable fuel, it can effectively use vitamin C. Of course, in so doing, your body only converts vitamin C back into refined, white sugar – without the associated vitamins [mostly B complex] and minerals found in natural sources of sugar.
Experiments have shown that taking massive doses of vitamin C for healing, etc. can be wasteful, except when fighting Ebola or cancer, as your body is only able to absorb a maximum of 200 mg at a time as it apparently converts some of the excess back to sugar and expels the rest.
The recent discovery of Liposomal vitamin C taken orally is also mysterious as it virtually forces itself into your body’s cells at approximately 10 times the rate achieved by injection of vitamin C directly into your bloodstream, which seems impossible!
This may come in handy as world class medical scientists have discovered that the Ebola virus wreaks havoc by consuming 100% of a person’s vitamin C which produces the same symptoms as scurvy and results in death. The Ebola virus converts our vitamin C back into sugar and eats it – ALL! This is very abnormal as the human body retains at least some vitamin C in virtually all other cases!
The antidote/cure is to consume massive amounts of vitamin C – up to 448,000 mg [one pound] several times a day to counteract the symptoms and allow it to perform its function as part of the body’s defense system and eat the virus before it has time to convert all the vitamin C back into sugar. Your body still tries to keep the vitamin C level down to 200 mg but most of the excess destroys the virus before it gets converted back to sugar.
Furthermore, studies have revealed that blood sugar levels in diabetics can get down as low as the 20s while taking liposomal vitamin C orally with no adverse effects until the vitamin C blood serum levels also get low. Apparently the liposomal vitamin C is crossing the blood-brain barrier and being converted back to refined, white sugar – avoiding diabetic shock or a coma. This suggests that Diabetics possibly could carry liposomal C instead of sugary sweets to relieve a low blood sugar spell although that idea requires further study. Regular Vitamin C may also work as a sugar substitute in a survival situation.
It’s also been shown that taking vitamin C orally before a glucose tolerance test can improve the results, so there appears to be a metabolic link between vitamin C and blood sugar tests.
Dr. Tom Levy made a video of the amazing effects of Liposomal C as follows:
An Australian Station [Ranch] Owner was brought back from death’s door with liposomal vitamin C as follows:
Doctors had given up on him & disallowed vitamin C treatment and a court order forced their hand.
Here’s how to make it yourself:
It also cures cancer according to the following:
According to Wikipedia, Vitamin C or L-ascorbic acid, C6H8O6 or simply ascorbate (the anion of ascorbic acid), is an essential nutrient for humans and certain other animal species. Vitamin C refers to a number of vitamers that have vitamin C activity in animals, including ascorbic acid and its salts, and some oxidized forms of the molecule like dehydroascorbic acid. Ascorbate and ascorbic acid are both naturally present in the body when either of these is introduced into cells, since the forms interconvert according to pH.
Author’s Note: Dog’s bodies manufacture their own vitamin C as needed to fight disease & maintain good health.
Vitamin C is a cofactor in at least eight enzymatic reactions, including several collagen [https://en.wikipedia.org/wiki/Collagen] synthesis reactions that, when dysfunctional, cause the most severe symptoms of scurvy. In animals, these reactions are especially important in wound-healing and in preventing bleeding from capillaries. It’s also effective as a nosebleed treatment in humans.
Ascorbate may also act as an antioxidant against oxidative stress. However, the fact that the enantiomer D-ascorbate (not found in nature) has identical antioxidant activity to L-ascorbate, yet far less vitamin activity, underscores the fact that most of the function of L-ascorbate as a vitamin relies not on its antioxidant properties, but upon enzymic reactions that are stereospecific. “Ascorbate” without the letter for the enantiomeric form is always presumed to be the chemical L-ascorbate.
Ascorbate (the anion of ascorbic acid) is required for a range of essential metabolic reactions in all animals and plants. It is made internally by almost all organisms; the main exceptions are most bats, all guinea pigs, capybaras, and the Anthropoidea (i.e., Haplorrhini, one of the two major primate suborders, consisting of tarsiers, monkeys, and humans and other apes). Ascorbate is also not synthesized by some species of birds and fish. All species that do not synthesize ascorbate require it in the diet. Deficiency in this vitamin causes the disease scurvy in humans.
The name ‘vitamin C’ always refers to the L-enantiomer of ascorbic acid and its oxidized forms. The opposite D-enantiomercalled D-ascorbate has equal antioxidant power, but is not found in nature, and has no physiological significance. When D-ascorbate is synthesized and given to animals that require vitamin C in their diets, it has been found to have far less vitamin activity than the L-enantiomer. Therefore, unless written otherwise, “ascorbate” and “ascorbic acid” refer in the nutritional literature to L-ascorbate and L-ascorbic acid, respectively. This notation will be followed in this article. Similarly, their oxidized derivatives (dehydroascorbate, etc., see below) are all L-enantiomers, and also need not be written with full sterochemical notation here.
Ascorbic acid is a weak sugar acid structurally related to glucose. In biological systems, ascorbic acid can be found only at low pH, but in neutral solutions above pH 5 is predominantly found in the ionized form, ascorbate. All of these molecules have vitamin C activity, therefore, and are used synonymously with vitamin C, unless otherwise specified.
The biological role of ascorbate is to act as a reducing agent, donating electrons to various enzymatic and a few non-enzymatic reactions. The one- and two-electron oxidized forms of vitamin C, semidehydroascorbic acid and dehydroascorbic acid, respectively, can be reduced in the body by glutathione and NADPH-dependent enzymatic mechanisms. The presence of glutathione in cells and extracellular fluids helps maintain ascorbate in a reduced state.
The vast majority of animals and plants are able to synthesize vitamin C, through a sequence of enzyme-driven steps, which convert monosaccharides to vitamin C. In plants, this is accomplished through the conversion of mannose or galactose to ascorbic acid. In some animals, glucose needed to produce ascorbate in the liver (in mammals and perching birds) is extracted from glycogen; ascorbate synthesis is a glycogenolysis-dependent process. In reptiles and birds the biosynthesis is carried out in the kidneys.
Among the animals that have lost the ability to synthesize vitamin C are simians and tarsiers, which together make up one of two major primate suborders, Haplorrhini. This group includes humans. The other more primitive primates (Strepsirrhini) have the ability to make vitamin C. Synthesis does not occur in a number of species (perhaps all species) in the small rodent family Caviidae that includes guinea pigs and capybaras, but occurs in other rodents (rats and mice do not need vitamin C in their diet, for example).
A number of species of passerine birds also do not synthesize, but not all of them, and those that do not are not clearly related; there is a theory that the ability was lost separately a number of times in birds. In particular, the ability to synthesize vitamin C is presumed to have been lost and then later re-acquired in at least two cases.
All tested families of bats, including major insect and fruit-eating bat families, cannot synthesize vitamin C. A trace of gulonolactone oxidase (GULO) was detected in only 1 of 34 bat species tested, across the range of 6 families of bats tested. However, recent results show that there are at least two species of bats, frugivorous bat (Rousettus leschenaultii) and insectivorous bat (Hipposideros armiger), that retain their ability of vitamin C production. The ability to synthesize vitamin C has also been lost in teleost fish.
These animals all lack the L-gulonolactone oxidase (GULO) enzyme, which is required in the last step of vitamin C synthesis, because they have a differing non-synthesizing gene for the enzyme (Pseudogene ΨGULO). A similar non-functional gene is present in the genome of the guinea pigs and in primates, including humans. Some of these species (including humans) are able to make do with the lower levels available from their diets by recycling oxidised vitamin C.
Most simians consume the vitamin in amounts 10 to 20 times higher than that recommended by governments for humans. This discrepancy constitutes much of the basis of the controversy on current recommended dietary allowances. It is countered by arguments that humans are very good at conserving dietary vitamin C, and are able to maintain blood levels of vitamin C comparable with other simians, on a far smaller dietary intake.
Like plants and animals, some microorganisms such as the yeast Saccharomyces cerevisiae have been shown to be able to synthesize vitamin C from simple sugars.
US Navy pilot (retired)
*This article is not intended as a substitute for medical advice